"0"^^ . . "3"^^ . . "1"^^ . . "angiotensin;deficitn\u00ED;hypertenze;inhibitor;my\u0161i;receptor;renovaskul\u00E1rn\u00ED"@en . . . "2004-03-23+01:00"^^ . "my\u0161i" . . "\u00DAloha renin-angiotensinov\u00E9ho syst\u00E9mu v regulaci ren\u00E1ln\u00EDch funkc\u00ED a krevn\u00EDho tlaku: studie na %22knock-out%22 my\u0161\u00EDch s absenc\u00ED receptoru AT1A." . "\u0158e\u0161en\u00ED projektu splnilo anotovan\u00E9 c\u00EDle. Jde o d\u016Fle\u017Eitou experiment\u00E1ln\u00ED pr\u00E1ci s v\u00FDznamn\u00FDmi implikacemi pro kliniku. Realiza\u010Dn\u00ED v\u00FDstupy pr\u00E1ce pom\u00E1haj\u00ED ke zlep\u0161en\u00ED identifikace etiologie art. hypertenze. N\u00E1vrh na Cenu ministra."@cs . "http://www.isvav.cz/projectDetail.do?rowId=NA5291"^^ . . "Role of the renin-angiotensin system in the control of renal function and blood pressure: studies in the %22knock-out%22 mouse with AT1A receptor absence."@en . "In mice with genetically evoked absence of AT-1 receptor for angiotensin II, which cannot respond to own endogenous angiotensin, the role of this autacoid for development and maintenance of blood pressure and renal function will be studied."@en . "receptor" . . "U my\u0161\u00ED s geneticky vyvolanou absenc\u00ED receptoru AT-1 pro AngII, kter\u00E9 nemohou reagovat na vlastn\u00ED angiotensin, bude zaji\u0161\u0165ov\u00E1n v\u00FDznam tohoto p\u016Fsobku jak pro norm\u00E1ln\u00ED regulaci krevn\u00EDho tlaku a ren\u00E1ln\u00EDch funkc\u00ED, tak pro rozvoj renovaskul\u00E1rn\u00ED hypertenze." . . . "hypertenze" . . "inhibitor" . . . . . "0"^^ . "NA5291" . "deficitn\u00ED" . "3"^^ . "angiotensin" .