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Statements

Subject Item
n2:RIV%2F67985823%3A_____%2F11%3A00372591%21RIV12-AV0-67985823
rdf:type
n13:Vysledek skos:Concept
dcterms:description
The cytokine tumor necrosis factor a (TNFa) is an established pain modulator in both the peripheral and central nervous systems. Modulation of nociceptive synaptic transmission in the spinal cord dorsal horn (DH) is thought to be involved in the development and maintenance of several pathological pain states. Increased levels of TNFa and its receptors (TNFR) in dorsal root ganglion (DRG) cells and in the spinal cord DH have been shown to play an essential role in neuropathic pain processing. In the present experiments the effect of TNFa incubation on modulation of primary afferent synaptic activity was investigated in a model of peripheral neuropathy The cytokine tumor necrosis factor a (TNFa) is an established pain modulator in both the peripheral and central nervous systems. Modulation of nociceptive synaptic transmission in the spinal cord dorsal horn (DH) is thought to be involved in the development and maintenance of several pathological pain states. Increased levels of TNFa and its receptors (TNFR) in dorsal root ganglion (DRG) cells and in the spinal cord DH have been shown to play an essential role in neuropathic pain processing. In the present experiments the effect of TNFa incubation on modulation of primary afferent synaptic activity was investigated in a model of peripheral neuropathy
dcterms:title
Modulation of spinal cord synaptic activity by tumor necrosis factor α in a model of peripheral neuropathy Modulation of spinal cord synaptic activity by tumor necrosis factor α in a model of peripheral neuropathy
skos:prefLabel
Modulation of spinal cord synaptic activity by tumor necrosis factor α in a model of peripheral neuropathy Modulation of spinal cord synaptic activity by tumor necrosis factor α in a model of peripheral neuropathy
skos:notation
RIV/67985823:_____/11:00372591!RIV12-AV0-67985823
n13:predkladatel
n18:ico%3A67985823
n3:aktivita
n12:P n12:Z
n3:aktivity
P(GA305/09/1228), P(LC554), Z(AV0Z50110509)
n3:cisloPeriodika
-
n3:dodaniDat
n11:2012
n3:domaciTvurceVysledku
n9:8912955 n9:7486774 n9:3703495
n3:druhVysledku
n4:J
n3:duvernostUdaju
n20:S
n3:entitaPredkladatele
n6:predkladatel
n3:idSjednocenehoVysledku
213435
n3:idVysledku
RIV/67985823:_____/11:00372591
n3:jazykVysledku
n17:eng
n3:klicovaSlova
TNF; neuropathy; pain
n3:klicoveSlovo
n8:pain n8:neuropathy n8:TNF
n3:kodStatuVydavatele
GB - Spojené království Velké Británie a Severního Irska
n3:kontrolniKodProRIV
[4AB4B08E3293]
n3:nazevZdroje
Journal of Neuroinflammation
n3:obor
n15:FH
n3:pocetDomacichTvurcuVysledku
3
n3:pocetTvurcuVysledku
3
n3:projekt
n10:GA305%2F09%2F1228 n10:LC554
n3:rokUplatneniVysledku
n11:2011
n3:svazekPeriodika
8
n3:tvurceVysledku
Paleček, Jiří Špicarová, Diana Nerandžič, Vladimír
n3:wos
000300341700001
n3:zamer
n7:AV0Z50110509
s:issn
1742-2094
s:numberOfPages
9
n16:doi
10.1186/1742-2094-8-177