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Statements

Subject Item
n2:RIV%2F62157124%3A16170%2F13%3A43871908%21RIV14-MSM-16170___
rdf:type
n8:Vysledek skos:Concept
dcterms:description
Soil contamination by lead-based paint sanded off the steel aviary resulted in poisoning of eight Cinereous and two Egyptian Vultures. A male Egyptian Vulture developed signs of apathy, polydipsia, polyuria, regurgitation, and stupor, and died on the next day. Liver, kidney and blood lead concentrations were 12.2, 8.16 and 2.66 ug/g, respectively. Blood Pb levels of Pb-exposed Cinereous Vultures were 1.571 +/- 0.510 ug/g shortly after intoxication, decreased to 0.530 +/- 0.165 ug/g without any therapy in a month and to 0.254 +/- 0.097 ug/g one month after CaNa2EDTA administration. Eight months later, blood lead levels decreased to close to the background of the control group. Blood parameters of healthy Pb-non-exposed Cinereous Vultures were compared with those of the exposed group prior to and after chelation therapy. Iron levels in the lead-exposed pre-treatment birds significantly decreased after chelation. Haematocrit levels in Pb-exposed birds were significantly lower than those of the controls and improved one month after chelation. Creatine kinase was higher in pre-treatment birds than in the controls but normalised after therapy. Alkaline phosphatase increased after chelation. A marked increase in the level of lipid peroxidation measured as thiobarbituric acid reactive species was demonstrated in birds both prior to and after chelation. The ferric reducing antioxidant power was significantly lower in pre-treatment vultures and returned to normal following chelation therapy. Blood metallothionein levels in lead-exposed birds were higher than in controls. Reduced glutathione dropped after CaNa2EDTA therapy, while oxidised glutathione was significantly lower in both pre- and post-treatment birds. A chick in an egg produced by a Cinereous Vulture female two months after lead toxicosis died on day 40 of artificial incubation. Lead concentrations in foetal tissues were consistent with levels causing avian mortality. Soil contamination by lead-based paint sanded off the steel aviary resulted in poisoning of eight Cinereous and two Egyptian Vultures. A male Egyptian Vulture developed signs of apathy, polydipsia, polyuria, regurgitation, and stupor, and died on the next day. Liver, kidney and blood lead concentrations were 12.2, 8.16 and 2.66 ug/g, respectively. Blood Pb levels of Pb-exposed Cinereous Vultures were 1.571 +/- 0.510 ug/g shortly after intoxication, decreased to 0.530 +/- 0.165 ug/g without any therapy in a month and to 0.254 +/- 0.097 ug/g one month after CaNa2EDTA administration. Eight months later, blood lead levels decreased to close to the background of the control group. Blood parameters of healthy Pb-non-exposed Cinereous Vultures were compared with those of the exposed group prior to and after chelation therapy. Iron levels in the lead-exposed pre-treatment birds significantly decreased after chelation. Haematocrit levels in Pb-exposed birds were significantly lower than those of the controls and improved one month after chelation. Creatine kinase was higher in pre-treatment birds than in the controls but normalised after therapy. Alkaline phosphatase increased after chelation. A marked increase in the level of lipid peroxidation measured as thiobarbituric acid reactive species was demonstrated in birds both prior to and after chelation. The ferric reducing antioxidant power was significantly lower in pre-treatment vultures and returned to normal following chelation therapy. Blood metallothionein levels in lead-exposed birds were higher than in controls. Reduced glutathione dropped after CaNa2EDTA therapy, while oxidised glutathione was significantly lower in both pre- and post-treatment birds. A chick in an egg produced by a Cinereous Vulture female two months after lead toxicosis died on day 40 of artificial incubation. Lead concentrations in foetal tissues were consistent with levels causing avian mortality.
dcterms:title
Lead toxicosis of captive vultures: Case description and responses to chelation therapy Lead toxicosis of captive vultures: Case description and responses to chelation therapy
skos:prefLabel
Lead toxicosis of captive vultures: Case description and responses to chelation therapy Lead toxicosis of captive vultures: Case description and responses to chelation therapy
skos:notation
RIV/62157124:16170/13:43871908!RIV14-MSM-16170___
n8:predkladatel
n19:orjk%3A16170
n3:aktivita
n5:S n5:I
n3:aktivity
I, S
n3:cisloPeriodika
11
n3:dodaniDat
n13:2014
n3:domaciTvurceVysledku
n11:8370346
n3:druhVysledku
n18:J
n3:duvernostUdaju
n16:S
n3:entitaPredkladatele
n14:predkladatel
n3:idSjednocenehoVysledku
84485
n3:idVysledku
RIV/62157124:16170/13:43871908
n3:jazykVysledku
n7:eng
n3:klicovaSlova
Treatment; Reproduction impairment; Oxidative stress; Biochemistry; Haematology; Plumbism; Neophron percnopterus; Aegypius monachus
n3:klicoveSlovo
n10:Neophron%20percnopterus n10:Aegypius%20monachus n10:Oxidative%20stress n10:Biochemistry n10:Haematology n10:Plumbism n10:Treatment n10:Reproduction%20impairment
n3:kodStatuVydavatele
GB - Spojené království Velké Británie a Severního Irska
n3:kontrolniKodProRIV
[3283BD36A939]
n3:nazevZdroje
BMC Veterinary Research
n3:obor
n4:GJ
n3:pocetDomacichTvurcuVysledku
1
n3:pocetTvurcuVysledku
15
n3:rokUplatneniVysledku
n13:2013
n3:svazekPeriodika
9
n3:tvurceVysledku
Osičková, Jitka Kizek, René Pikula, Jiří Škochová, Hana Hájková, Pavlína Banďouchová, Hana Adam, Vojtěch Sobotka, Jakub Král, Jiří Sedláčková, Jana Bednářová, Ivana Beklová, Miroslava Treml, František Pohanka, Miroslav Ondráček, Karel
n3:wos
000314925800001
s:issn
1746-6148
s:numberOfPages
10
n6:doi
10.1186/1746-6148-9-11
n9:organizacniJednotka
16170