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Statements

Subject Item
n2:RIV%2F00216224%3A14110%2F13%3A00069377%21RIV14-MSM-14110___
rdf:type
n16:Vysledek skos:Concept
dcterms:description
Brugada syndrome (BrS), an inherited arrhythmogenic disease first described in 1992, is characterized by ST segment elevations on the electrocardiogram in the right precordium and by a high occurrence of arrhythmias including the life-threatening ventricular tachycardia/fibrillation. Knowledge of the underlying mechanisms of formation of arrhythmogenic substrate in BrS is essential, namely for the risk stratification of BrS patients and their therapy which is still restrained almost exclusively to the implantation of cardioverter/defibrillator. In spite of many crucial findings in this field published within recent years, the final consistent view has not been established so far. Hence, BrS described 20 years ago remains an actual topic of both clinical and experimental studies. Brugada syndrome (BrS), an inherited arrhythmogenic disease first described in 1992, is characterized by ST segment elevations on the electrocardiogram in the right precordium and by a high occurrence of arrhythmias including the life-threatening ventricular tachycardia/fibrillation. Knowledge of the underlying mechanisms of formation of arrhythmogenic substrate in BrS is essential, namely for the risk stratification of BrS patients and their therapy which is still restrained almost exclusively to the implantation of cardioverter/defibrillator. In spite of many crucial findings in this field published within recent years, the final consistent view has not been established so far. Hence, BrS described 20 years ago remains an actual topic of both clinical and experimental studies.
dcterms:title
Arrhythmogenesis in Brugada syndrome: Impact and constrains of current concepts Arrhythmogenesis in Brugada syndrome: Impact and constrains of current concepts
skos:prefLabel
Arrhythmogenesis in Brugada syndrome: Impact and constrains of current concepts Arrhythmogenesis in Brugada syndrome: Impact and constrains of current concepts
skos:notation
RIV/00216224:14110/13:00069377!RIV14-MSM-14110___
n16:predkladatel
n17:orjk%3A14110
n3:aktivita
n6:Z
n3:aktivity
Z(MSM0021622402)
n3:cisloPeriodika
5
n3:dodaniDat
n18:2014
n3:domaciTvurceVysledku
n14:1457160
n3:druhVysledku
n12:J
n3:duvernostUdaju
n20:S
n3:entitaPredkladatele
n15:predkladatel
n3:idSjednocenehoVysledku
62098
n3:idVysledku
RIV/00216224:14110/13:00069377
n3:jazykVysledku
n11:eng
n3:klicovaSlova
Arrhythmogenesis; Atrial fibrillation; Brugada syndrome; Discontinuous conduction; Dysfunction; Mutation
n3:klicoveSlovo
n4:Dysfunction n4:Arrhythmogenesis n4:Atrial%20fibrillation n4:Mutation n4:Discontinuous%20conduction n4:Brugada%20syndrome
n3:kodStatuVydavatele
NL - Nizozemsko
n3:kontrolniKodProRIV
[761410D05C8B]
n3:nazevZdroje
International Journal of Cardiology
n3:obor
n9:FA
n3:pocetDomacichTvurcuVysledku
1
n3:pocetTvurcuVysledku
1
n3:rokUplatneniVysledku
n18:2013
n3:svazekPeriodika
167
n3:tvurceVysledku
Bébarová, Markéta
n3:wos
000323569600029
n3:zamer
n13:MSM0021622402
s:issn
0167-5273
s:numberOfPages
12
n8:doi
10.1016/j.ijcard.2012.12.019
n19:organizacniJednotka
14110