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Statements

Subject Item
n2:RIV%2F00216208%3A11510%2F14%3A10287685%21RIV15-MSM-11510___
rdf:type
skos:Concept n14:Vysledek
dcterms:description
Intracranial hypertension represents a significant risk factor of poor neurological outcome in critically ill, specifically those with craniotrauma. Methods: In a porcine model of cardiac arrest (CA) the dynamics of intracranial pressure (ICP) was studied. Threeminutes of untreated CA were followed by 5-minutes of cardiopulmonary resuscitation (CPR) with chest compressions and mechanical ventilation. No other interventions were allowed since out-of-hospital resuscitation was simulated. Throughout the protocol the ICP, arterial and venous pressures and carotid blood flow were continuously recorded. Results: During cardiac arrest ICP increased to 27 +/- 4.8 mmHg, i.e. hypertensive values. Mechanical chest compressions resulted in a transient ICP increase followed by a steady decline at approximately 2.25 mmHg/min. By five minutes of CPR intracranial pressure reached prearrest values despite significant elevation of central venous pressure (39 mmHg) and gradual decrease of arterial pressure and flow. We hypothesize that cerebral autoregulation is capable of maintaining ICP even during failing circulation. Intracranial hypertension represents a significant risk factor of poor neurological outcome in critically ill, specifically those with craniotrauma. Methods: In a porcine model of cardiac arrest (CA) the dynamics of intracranial pressure (ICP) was studied. Threeminutes of untreated CA were followed by 5-minutes of cardiopulmonary resuscitation (CPR) with chest compressions and mechanical ventilation. No other interventions were allowed since out-of-hospital resuscitation was simulated. Throughout the protocol the ICP, arterial and venous pressures and carotid blood flow were continuously recorded. Results: During cardiac arrest ICP increased to 27 +/- 4.8 mmHg, i.e. hypertensive values. Mechanical chest compressions resulted in a transient ICP increase followed by a steady decline at approximately 2.25 mmHg/min. By five minutes of CPR intracranial pressure reached prearrest values despite significant elevation of central venous pressure (39 mmHg) and gradual decrease of arterial pressure and flow. We hypothesize that cerebral autoregulation is capable of maintaining ICP even during failing circulation.
dcterms:title
Intracranial Pressure Declines During Cardiopulmonary Resuscitation in Animal Model Intracranial Pressure Declines During Cardiopulmonary Resuscitation in Animal Model
skos:prefLabel
Intracranial Pressure Declines During Cardiopulmonary Resuscitation in Animal Model Intracranial Pressure Declines During Cardiopulmonary Resuscitation in Animal Model
skos:notation
RIV/00216208:11510/14:10287685!RIV15-MSM-11510___
n5:aktivita
n7:S n7:P n7:I
n5:aktivity
I, P(GAP407/10/1624), S
n5:cisloPeriodika
S1
n5:dodaniDat
n11:2015
n5:domaciTvurceVysledku
n8:6404626
n5:druhVysledku
n18:J
n5:duvernostUdaju
n13:S
n5:entitaPredkladatele
n10:predkladatel
n5:idSjednocenehoVysledku
22597
n5:idVysledku
RIV/00216208:11510/14:10287685
n5:jazykVysledku
n15:eng
n5:klicovaSlova
Model.; Animal; Resuscitation; Cardiopulmonary; During; Declines; Pressure; Intracranial
n5:klicoveSlovo
n9:Resuscitation n9:Pressure n9:Intracranial n9:Declines n9:Animal n9:Cardiopulmonary n9:Model. n9:During
n5:kodStatuVydavatele
DE - Spolková republika Německo
n5:kontrolniKodProRIV
[88CF8328DF53]
n5:nazevZdroje
Biomedizinische Technik. Biomedical engineering
n5:obor
n17:BO
n5:pocetDomacichTvurcuVysledku
1
n5:pocetTvurcuVysledku
3
n5:projekt
n6:GAP407%2F10%2F1624
n5:rokUplatneniVysledku
n11:2014
n5:svazekPeriodika
59
n5:tvurceVysledku
Hrachovina, Matěj Mlček, Mikuláš Mayer, Martin
n5:wos
000350405700430
s:issn
0013-5585
s:numberOfPages
3
n12:organizacniJednotka
11510