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Statements

Subject Item
n2:RIV%2F00216208%3A11150%2F13%3A10194859%21RIV14-MSM-11150___
rdf:type
skos:Concept n16:Vysledek
rdfs:seeAlso
http://dx.doi.org/10.1016/j.mvr.2012.10.002
dcterms:description
The present study suggests that the intestinal microcirculation in experimental sepsis can be modulated by estradiol receptor agonists.These findings might explain the relative resistance of the female gastrointestinal barrier function to systemic inflammation.Estradiol receptors agonists represent a novel alternative for experimental treatment with the non-selective steroid hormone estradiol in sepsis.However,further studies are needed to provide additional,mechanistic information to understand the effects of estradiol receptor agonists.The use of such agonists would be beneficial as it might avoid the side effects reported after estradiol hormone therapy. The present study suggests that the intestinal microcirculation in experimental sepsis can be modulated by estradiol receptor agonists.These findings might explain the relative resistance of the female gastrointestinal barrier function to systemic inflammation.Estradiol receptors agonists represent a novel alternative for experimental treatment with the non-selective steroid hormone estradiol in sepsis.However,further studies are needed to provide additional,mechanistic information to understand the effects of estradiol receptor agonists.The use of such agonists would be beneficial as it might avoid the side effects reported after estradiol hormone therapy.
dcterms:title
Estradiol receptors agonists induced effects in rat intestinal microcirculation during sepsis Estradiol receptors agonists induced effects in rat intestinal microcirculation during sepsis
skos:prefLabel
Estradiol receptors agonists induced effects in rat intestinal microcirculation during sepsis Estradiol receptors agonists induced effects in rat intestinal microcirculation during sepsis
skos:notation
RIV/00216208:11150/13:10194859!RIV14-MSM-11150___
n16:predkladatel
n19:orjk%3A11150
n3:aktivita
n11:I
n3:aktivity
I
n3:cisloPeriodika
January 2013
n3:dodaniDat
n9:2014
n3:domaciTvurceVysledku
n17:2920387
n3:druhVysledku
n13:J
n3:duvernostUdaju
n7:S
n3:entitaPredkladatele
n18:predkladatel
n3:idSjednocenehoVysledku
73310
n3:idVysledku
RIV/00216208:11150/13:10194859
n3:jazykVysledku
n15:eng
n3:klicovaSlova
lung injury; cecal ligation; inflammatory response; bacterial translocation; blood-flow; polymicrobial sepsis; nitric-oxide; trauma-hemorrhage; estrogen-receptor; ascendens stent peritonitis
n3:klicoveSlovo
n4:inflammatory%20response n4:lung%20injury n4:estrogen-receptor n4:bacterial%20translocation n4:ascendens%20stent%20peritonitis n4:blood-flow n4:nitric-oxide n4:polymicrobial%20sepsis n4:cecal%20ligation n4:trauma-hemorrhage
n3:kodStatuVydavatele
US - Spojené státy americké
n3:kontrolniKodProRIV
[CDFF5111E6B4]
n3:nazevZdroje
Microvascular Research
n3:obor
n20:FP
n3:pocetDomacichTvurcuVysledku
1
n3:pocetTvurcuVysledku
9
n3:rokUplatneniVysledku
n9:2013
n3:svazekPeriodika
85
n3:tvurceVysledku
Sharawy, Nivin Dombrowski, Frank Ribback, Silvia Lehmann, Christian Kern, Hartmut Černý, Vladimír Wendt, Michael Pavlovic, Dragan Al-Banna, Nadia
n3:wos
000314318800017
s:issn
0026-2862
s:numberOfPages
10
n14:doi
10.1016/j.mvr.2012.10.002
n12:organizacniJednotka
11150