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Statements

Subject Item
n2:RIV%2F00216208%3A11150%2F13%3A10139197%21RIV14-MSM-11150___
rdf:type
skos:Concept n16:Vysledek
rdfs:seeAlso
http://prf.sagepub.com/content/28/5/377
dcterms:description
Coronary artery bypass grafting (CABG) is performed with the use of cardiopulmonary bypass (CPB) and cardioplegic arrest (CA) of the heart. The advantage of this technique, alternatively referred to as %22on-pump%22 surgery, resides, for the surgeon, in relatively easy access to and manipulation with the non-beating, bloodless heart. However, the advantage that is, thereby, gained by the patient is paid off by an increased susceptibility to postoperative systemic inflammatory response syndrome (SIRS). Under unfavorable conditions, the inflammatory syndrome may develop into life-threatening forms of MODS (multiple organ dysfunction syndrome) or even MOFS (multiple organ failure syndrome). Deliberate avoidance of CPB, also known as %22off-pump%22 surgery, attenuates early postoperative inflammation throughout its trajectory of SIRSRIGHTWARDS ARROWMODSRIGHTWARDS ARROWMOFS, but, in the long run, there appears to be no substantial difference in the overall mortality rates. In the last years, our knowledge of the pathophysiology of surgical inflammation has increased considerably. Recent findings, highlighting the as yet rather obscure role of pentraxin 3 (PTX3) in these processes, are discussed in this review article. Coronary artery bypass grafting (CABG) is performed with the use of cardiopulmonary bypass (CPB) and cardioplegic arrest (CA) of the heart. The advantage of this technique, alternatively referred to as %22on-pump%22 surgery, resides, for the surgeon, in relatively easy access to and manipulation with the non-beating, bloodless heart. However, the advantage that is, thereby, gained by the patient is paid off by an increased susceptibility to postoperative systemic inflammatory response syndrome (SIRS). Under unfavorable conditions, the inflammatory syndrome may develop into life-threatening forms of MODS (multiple organ dysfunction syndrome) or even MOFS (multiple organ failure syndrome). Deliberate avoidance of CPB, also known as %22off-pump%22 surgery, attenuates early postoperative inflammation throughout its trajectory of SIRSRIGHTWARDS ARROWMODSRIGHTWARDS ARROWMOFS, but, in the long run, there appears to be no substantial difference in the overall mortality rates. In the last years, our knowledge of the pathophysiology of surgical inflammation has increased considerably. Recent findings, highlighting the as yet rather obscure role of pentraxin 3 (PTX3) in these processes, are discussed in this review article.
dcterms:title
The long pentraxin PTX3: a candidate anti-inflammatory mediator in cardiac surgery The long pentraxin PTX3: a candidate anti-inflammatory mediator in cardiac surgery
skos:prefLabel
The long pentraxin PTX3: a candidate anti-inflammatory mediator in cardiac surgery The long pentraxin PTX3: a candidate anti-inflammatory mediator in cardiac surgery
skos:notation
RIV/00216208:11150/13:10139197!RIV14-MSM-11150___
n16:predkladatel
n17:orjk%3A11150
n4:aktivita
n19:I
n4:aktivity
I
n4:cisloPeriodika
5
n4:dodaniDat
n7:2014
n4:domaciTvurceVysledku
n18:8222525 n18:6283322 n18:7813813 n18:1730452
n4:druhVysledku
n15:J
n4:duvernostUdaju
n11:S
n4:entitaPredkladatele
n12:predkladatel
n4:idSjednocenehoVysledku
85351
n4:idVysledku
RIV/00216208:11150/13:10139197
n4:jazykVysledku
n20:eng
n4:klicovaSlova
interleukin-10; pentraxin 3; alarmins; sterile inflammation; cardiopulmonary bypass; coronary artery bypass grafting
n4:klicoveSlovo
n6:interleukin-10 n6:pentraxin%203 n6:alarmins n6:sterile%20inflammation n6:cardiopulmonary%20bypass n6:coronary%20artery%20bypass%20grafting
n4:kodStatuVydavatele
GB - Spojené království Velké Británie a Severního Irska
n4:kontrolniKodProRIV
[C7BD407125B9]
n4:nazevZdroje
Perfusion
n4:obor
n9:FA
n4:pocetDomacichTvurcuVysledku
4
n4:pocetTvurcuVysledku
5
n4:rokUplatneniVysledku
n7:2013
n4:svazekPeriodika
28
n4:tvurceVysledku
Holubcová, Zdeňka Kuneš, Pavel Krejsek, Jan Koláčková, Martina Manďák, Jiří
n4:wos
000323311800002
s:issn
0267-6591
s:numberOfPages
13
n14:doi
10.1177/0267659113483799
n5:organizacniJednotka
11150