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Statements

Subject Item
n2:RIV%2F00216208%3A11130%2F10%3A6241%21RIV11-MZ0-11130___
rdf:type
skos:Concept n17:Vysledek
dcterms:description
Burkholderia cepacia complex (Bcc) bacteria have gained notoriety as pathogens in cystic fibrosis (CF) because they are difficult to identify and treat and have the ability to spread between CF individuals. Of the 17 formally named species within the complex, Burkholderia cenocepacia dominate in CF, with highly transmissible strains such as the ET-12 (ST-28) and Czech strain (ST-32). The majority of research on the molecular pathogenesis of Bcc bacteria has focused on B. cenocepacia ET-12 epidemic lineage, with gene mutation, genome sequence analysis and most recently global gene expression studies shedding considerable light on the virulence and antimicrobial resistance of this pathogen. These studies demonstrate that the ability of B. cenocepacia to acquire foreign DNA, regulate gene expression, compete for iron during infection, and mediate antimicrobial resistance via its polysaccharides, are key features that underpin the virulence of different strains. Burkholderia cepacia complex (Bcc) bacteria have gained notoriety as pathogens in cystic fibrosis (CF) because they are difficult to identify and treat and have the ability to spread between CF individuals. Of the 17 formally named species within the complex, Burkholderia cenocepacia dominate in CF, with highly transmissible strains such as the ET-12 (ST-28) and Czech strain (ST-32). The majority of research on the molecular pathogenesis of Bcc bacteria has focused on B. cenocepacia ET-12 epidemic lineage, with gene mutation, genome sequence analysis and most recently global gene expression studies shedding considerable light on the virulence and antimicrobial resistance of this pathogen. These studies demonstrate that the ability of B. cenocepacia to acquire foreign DNA, regulate gene expression, compete for iron during infection, and mediate antimicrobial resistance via its polysaccharides, are key features that underpin the virulence of different strains.
dcterms:title
Burkholderia cenocepacia in cystic fibrosis: epidemiology and molecular mechanisms of virulence Burkholderia cenocepacia in cystic fibrosis: epidemiology and molecular mechanisms of virulence
skos:prefLabel
Burkholderia cenocepacia in cystic fibrosis: epidemiology and molecular mechanisms of virulence Burkholderia cenocepacia in cystic fibrosis: epidemiology and molecular mechanisms of virulence
skos:notation
RIV/00216208:11130/10:6241!RIV11-MZ0-11130___
n3:aktivita
n8:P n8:Z
n3:aktivity
P(NS10543), Z(MSM0021620812)
n3:cisloPeriodika
7
n3:dodaniDat
n13:2011
n3:domaciTvurceVysledku
n9:7780710
n3:druhVysledku
n6:J
n3:duvernostUdaju
n19:S
n3:entitaPredkladatele
n15:predkladatel
n3:idSjednocenehoVysledku
249384
n3:idVysledku
RIV/00216208:11130/10:6241
n3:jazykVysledku
n10:eng
n3:klicovaSlova
Burkholderia cepacia; epidemiology; genomics; review; multilocus sequence typing; virulence factors; quorum-sensing system; cepacia-complex; genomovar III; ornibactin biosynthesis; global epidemiology; oxidative stress
n3:klicoveSlovo
n4:multilocus%20sequence%20typing n4:genomovar%20III n4:virulence%20factors n4:quorum-sensing%20system n4:global%20epidemiology n4:epidemiology n4:review n4:oxidative%20stress n4:Burkholderia%20cepacia n4:genomics n4:ornibactin%20biosynthesis n4:cepacia-complex
n3:kodStatuVydavatele
GB - Spojené království Velké Británie a Severního Irska
n3:kontrolniKodProRIV
[D5DEDB8B5F5A]
n3:nazevZdroje
Clinical Microbiology and Infection
n3:obor
n14:EE
n3:pocetDomacichTvurcuVysledku
1
n3:pocetTvurcuVysledku
2
n3:projekt
n11:NS10543
n3:rokUplatneniVysledku
n13:2010
n3:svazekPeriodika
16
n3:tvurceVysledku
Mahenthiralingam, E. Dřevínek, Pavel
n3:wos
000278645700004
n3:zamer
n16:MSM0021620812
s:issn
1198-743X
s:numberOfPages
10
n18:organizacniJednotka
11130