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Statements

Subject Item
n2:RIV%2F00216208%3A11110%2F13%3A00011383%21RIV14-MSM-11110___
rdf:type
n11:Vysledek skos:Concept
rdfs:seeAlso
http://dx.doi.org/10.1007/978-1-4614-5203-4_3
dcterms:description
Like every muscle in the body, the heart adapts its structure and mass to functional requirements, which are primarily determined by loading conditions. These can be classified as pressure loading (increased afterload, as in hypertension or banding of an artery), or volume loading (preload, which could be increased by creating an arterio-venous shunt, valve regurgitation, or shifting the blood streams). While numerous models exist for modifying the loading conditions postnatally, this chapter focuses on those few created in the developing heart, chiefly during prenatal development. As the reactions of the early postnatal heart are somewhat similar to the response of the fetal one, discussion of early postnatal models is also included. Principal findings from these studies are considered in the light of clinical scenarios of abnormal loading in congenital heart malformations and their optimal surgical management. Like every muscle in the body, the heart adapts its structure and mass to functional requirements, which are primarily determined by loading conditions. These can be classified as pressure loading (increased afterload, as in hypertension or banding of an artery), or volume loading (preload, which could be increased by creating an arterio-venous shunt, valve regurgitation, or shifting the blood streams). While numerous models exist for modifying the loading conditions postnatally, this chapter focuses on those few created in the developing heart, chiefly during prenatal development. As the reactions of the early postnatal heart are somewhat similar to the response of the fetal one, discussion of early postnatal models is also included. Principal findings from these studies are considered in the light of clinical scenarios of abnormal loading in congenital heart malformations and their optimal surgical management.
dcterms:title
Prenatal adaptations to overload Prenatal adaptations to overload
skos:prefLabel
Prenatal adaptations to overload Prenatal adaptations to overload
skos:notation
RIV/00216208:11110/13:00011383!RIV14-MSM-11110___
n11:predkladatel
n24:orjk%3A11110
n3:aktivita
n7:Z n7:I n7:P
n3:aktivity
I, P(GA304/08/0615), P(GAP302/11/1308), Z(AV0Z50110509), Z(MSM0021620806)
n3:dodaniDat
n17:2014
n3:domaciTvurceVysledku
n14:5754623 n14:7220014
n3:druhVysledku
n21:C
n3:duvernostUdaju
n5:S
n3:entitaPredkladatele
n13:predkladatel
n3:idSjednocenehoVysledku
98919
n3:idVysledku
RIV/00216208:11110/13:00011383
n3:jazykVysledku
n23:eng
n3:klicovaSlova
prenatal myocardium; left atrial ligation; conotruncal banding; pulmonary artery banding; aortic banding; hypertrophy; hyperplasia; fetal sheep; chick embryo
n3:klicoveSlovo
n12:hypertrophy n12:chick%20embryo n12:fetal%20sheep n12:pulmonary%20artery%20banding n12:hyperplasia n12:conotruncal%20banding n12:aortic%20banding n12:left%20atrial%20ligation n12:prenatal%20myocardium
n3:kontrolniKodProRIV
[540FDC608122]
n3:mistoVydani
New York
n3:nazevEdiceCisloSvazku
Advances in Biochemistry in Health and Disease, Vol 4
n3:nazevZdroje
Cardiac adaptations. Molecular mechanisms
n3:obor
n18:FA
n3:pocetDomacichTvurcuVysledku
2
n3:pocetStranKnihy
465
n3:pocetTvurcuVysledku
2
n3:projekt
n15:GA304%2F08%2F0615 n15:GAP302%2F11%2F1308
n3:rokUplatneniVysledku
n17:2013
n3:tvurceVysledku
Sedmera, David Peševski, Živorad
n3:zamer
n6:AV0Z50110509 n6:MSM0021620806
s:numberOfPages
17
n22:doi
10.1007/978-1-4614-5203-4_3
n19:hasPublisher
Springer-Verlag
n20:isbn
978-1-4614-5202-7
n9:organizacniJednotka
11110