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Statements

Subject Item
n2:RIV%2F00179906%3A_____%2F12%3A10125277%21RIV13-MZ0-00179906
rdf:type
n6:Vysledek skos:Concept
rdfs:seeAlso
http://www.sciencedirect.com/science/article/pii/S0344033812000453
dcterms:description
Calcific aortic disease (CAS) is considered a form of atherosclerosis and, like the latter, of inflammatory origin. The aim of our work was to study the pattern of cellular infiltrate in CAS. Fifteen operatively excised calcified aortic valves were examined by means of histology and immunohistochemistry (CD20, CD79α, CD3, CD4, CD8, CD68, CD138, BJK, BJL, IgA, IgD, IgG, IgG4, and IgM). The findings present CAS as a chronic inflammatory process with polyclonal infiltrates comprising lymphocytes, plasma cells and histiocytes. The inflammatory process was the same in both subsets of CAS- those with trileaflet (normally formed) valves and those with congenitally bicuspid valves. Calcific aortic disease (CAS) is considered a form of atherosclerosis and, like the latter, of inflammatory origin. The aim of our work was to study the pattern of cellular infiltrate in CAS. Fifteen operatively excised calcified aortic valves were examined by means of histology and immunohistochemistry (CD20, CD79α, CD3, CD4, CD8, CD68, CD138, BJK, BJL, IgA, IgD, IgG, IgG4, and IgM). The findings present CAS as a chronic inflammatory process with polyclonal infiltrates comprising lymphocytes, plasma cells and histiocytes. The inflammatory process was the same in both subsets of CAS- those with trileaflet (normally formed) valves and those with congenitally bicuspid valves.
dcterms:title
Calcific aortic stenosis: immunohistochemical analysis of inflammatory infiltrate Calcific aortic stenosis: immunohistochemical analysis of inflammatory infiltrate
skos:prefLabel
Calcific aortic stenosis: immunohistochemical analysis of inflammatory infiltrate Calcific aortic stenosis: immunohistochemical analysis of inflammatory infiltrate
skos:notation
RIV/00179906:_____/12:10125277!RIV13-MZ0-00179906
n6:predkladatel
n7:ico%3A00179906
n4:aktivita
n16:I
n4:aktivity
I
n4:cisloPeriodika
4
n4:dodaniDat
n13:2013
n4:domaciTvurceVysledku
n15:9951598 n15:1053132 n15:2536846 n15:1373668 n15:6953891
n4:druhVysledku
n19:J
n4:duvernostUdaju
n14:S
n4:entitaPredkladatele
n11:predkladatel
n4:idSjednocenehoVysledku
125792
n4:idVysledku
RIV/00179906:_____/12:10125277
n4:jazykVysledku
n8:eng
n4:klicovaSlova
sclerosing disease; Immunohistochemistry; Inflammation; Calcific aortic stenosis
n4:klicoveSlovo
n5:Immunohistochemistry n5:Inflammation n5:sclerosing%20disease n5:Calcific%20aortic%20stenosis
n4:kodStatuVydavatele
NL - Nizozemsko
n4:kontrolniKodProRIV
[223E1A72C9F4]
n4:nazevZdroje
Pathology Research and Practice
n4:obor
n10:FP
n4:pocetDomacichTvurcuVysledku
5
n4:pocetTvurcuVysledku
5
n4:rokUplatneniVysledku
n13:2012
n4:svazekPeriodika
208
n4:tvurceVysledku
Laco, Jan Šteiner, Ivo Rozkoš, Tomáš Harrer, Jan Krbal, Lukáš
n4:wos
000303850000007
s:issn
0344-0338
s:numberOfPages
4
n17:doi
10.1016/j.prp.2012.02.009