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Statements

Subject Item
n2:RIV%2F00159816%3A_____%2F12%3A%230001023%21RIV13-MZ0-00159816
rdf:type
skos:Concept n16:Vysledek
dcterms:description
There is substantial evidence in the literature that the basal ganglia (BG), namely the striatum and pallidum, are involved in temporal lobe epilepsy (TLE). The BG are probably not involved in elaborating clinical seizures, as they do not produce specific epileptiform activity and there is no evident change in the electrical activity in the BG immediately after seizure onset. The data we obtained by direct ictal recording in the BG [1,2], as well as a large body of experimental and clinical evidence reported by other groups, suggest an inhibitory role of the BG during temporal lobe seizures. The BG may have a remote influence on cortical oscillatory processes related to control of epileptic seizures via their feedback pathways to the cortex. (C) 2012 Elsevier Inc. All rights reserved. There is substantial evidence in the literature that the basal ganglia (BG), namely the striatum and pallidum, are involved in temporal lobe epilepsy (TLE). The BG are probably not involved in elaborating clinical seizures, as they do not produce specific epileptiform activity and there is no evident change in the electrical activity in the BG immediately after seizure onset. The data we obtained by direct ictal recording in the BG [1,2], as well as a large body of experimental and clinical evidence reported by other groups, suggest an inhibitory role of the BG during temporal lobe seizures. The BG may have a remote influence on cortical oscillatory processes related to control of epileptic seizures via their feedback pathways to the cortex. (C) 2012 Elsevier Inc. All rights reserved.
dcterms:title
Do the basal ganglia inhibit seizure activity in temporal lobe epilepsy? Do the basal ganglia inhibit seizure activity in temporal lobe epilepsy?
skos:prefLabel
Do the basal ganglia inhibit seizure activity in temporal lobe epilepsy? Do the basal ganglia inhibit seizure activity in temporal lobe epilepsy?
skos:notation
RIV/00159816:_____/12:#0001023!RIV13-MZ0-00159816
n16:predkladatel
n17:ico%3A00159816
n3:aktivita
n15:I n15:P
n3:aktivity
I, P(ED1.1.00/02.0068)
n3:cisloPeriodika
1
n3:dodaniDat
n9:2013
n3:domaciTvurceVysledku
n8:9233075 n8:3864871 n8:1791656 n8:8132208
n3:druhVysledku
n14:J
n3:duvernostUdaju
n19:S
n3:entitaPredkladatele
n12:predkladatel
n3:idSjednocenehoVysledku
131790
n3:idVysledku
RIV/00159816:_____/12:#0001023
n3:jazykVysledku
n13:eng
n3:klicovaSlova
Basal ganglia, Temporal lobe, Epilepsy
n3:klicoveSlovo
n5:Epilepsy n5:Basal%20ganglia n5:Temporal%20lobe
n3:kodStatuVydavatele
US - Spojené státy americké
n3:kontrolniKodProRIV
[748EC1A35BAF]
n3:nazevZdroje
EPILEPSY & BEHAVIOR
n3:obor
n4:FH
n3:pocetDomacichTvurcuVysledku
4
n3:pocetTvurcuVysledku
4
n3:projekt
n11:ED1.1.00%2F02.0068
n3:rokUplatneniVysledku
n9:2012
n3:svazekPeriodika
25
n3:tvurceVysledku
Kuba, Robert Brázdil, Milan Rektor, Ivan Chrastina, Jan
s:issn
1525-5050
s:numberOfPages
4
n18:doi
10.1016/j.yebeh.2012.04.125