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  • Abscisic acid (ABA) is the key hormone in plant responses to water deficit, caused by abiotic stresses associated with dehydration (drought, salinity, and cold) and during specific developmental stages (e.g., seed maturation). ABA signal transduction pathway is initiated by binding of ABA to its receptors (GTG1, GTG2, Mg protoporphyrin chelatase H-subunit, and 14-member family of PYR/PYL/RCAR proteins). PYR/PYL/RCARs directly interact with ABA and the main negative regulators of ABA signaling - type-2C serine threonine protein phosphatases (PP2Cs). Formation of the complex results in derepression of specific protein kinases, especially of SnRK2 family. In cytoplasm, SnRK2.6 (OST1) modulates the activity of ion channels (stimulating anion efflux and inhibiting K+ influx). By fast stimulation of ion efflux, turgor of stomata cells is reduced, which results in their closure. In the nucleus, ABA signal transduction induces a wide change in the transcriptome not only by stimulation of the expression of defense genes, but also by degradation of specific mRNAs, by change of mRNA stability, splicing, and transport. Elucidation of the mechanism of ABA signal transduction significantly contributes to establishment of a suitable strategy for elevation of plant tolerance to abiotic stresses.
  • Abscisic acid (ABA) is the key hormone in plant responses to water deficit, caused by abiotic stresses associated with dehydration (drought, salinity, and cold) and during specific developmental stages (e.g., seed maturation). ABA signal transduction pathway is initiated by binding of ABA to its receptors (GTG1, GTG2, Mg protoporphyrin chelatase H-subunit, and 14-member family of PYR/PYL/RCAR proteins). PYR/PYL/RCARs directly interact with ABA and the main negative regulators of ABA signaling - type-2C serine threonine protein phosphatases (PP2Cs). Formation of the complex results in derepression of specific protein kinases, especially of SnRK2 family. In cytoplasm, SnRK2.6 (OST1) modulates the activity of ion channels (stimulating anion efflux and inhibiting K+ influx). By fast stimulation of ion efflux, turgor of stomata cells is reduced, which results in their closure. In the nucleus, ABA signal transduction induces a wide change in the transcriptome not only by stimulation of the expression of defense genes, but also by degradation of specific mRNAs, by change of mRNA stability, splicing, and transport. Elucidation of the mechanism of ABA signal transduction significantly contributes to establishment of a suitable strategy for elevation of plant tolerance to abiotic stresses. (en)
Title
  • Abscisic Acid Signaling in Plants
  • Abscisic Acid Signaling in Plants (en)
skos:prefLabel
  • Abscisic Acid Signaling in Plants
  • Abscisic Acid Signaling in Plants (en)
skos:notation
  • RIV/61389030:_____/12:00382532!RIV13-AV0-61389030
http://linked.open...avai/riv/aktivita
http://linked.open...avai/riv/aktivity
  • P(GA522/09/2058), Z(AV0Z50380511)
http://linked.open...vai/riv/dodaniDat
http://linked.open...aciTvurceVysledku
http://linked.open.../riv/druhVysledku
http://linked.open...iv/duvernostUdaju
http://linked.open...titaPredkladatele
http://linked.open...dnocenehoVysledku
  • 120785
http://linked.open...ai/riv/idVysledku
  • RIV/61389030:_____/12:00382532
http://linked.open...riv/jazykVysledku
http://linked.open.../riv/klicovaSlova
  • PP2C; PYR/PYL/RCAR proteins; Abscisic acid (en)
http://linked.open.../riv/klicoveSlovo
http://linked.open...ontrolniKodProRIV
  • [56E0342A1CF0]
http://linked.open...i/riv/mistoVydani
  • NEW YORK
http://linked.open...i/riv/nazevZdroje
  • ABIOTIC STRESS RESPONSES IN PLANTS: METABOLISM, PRODUCTIVITY AND SUSTAINABILITY
http://linked.open...in/vavai/riv/obor
http://linked.open...ichTvurcuVysledku
http://linked.open...v/pocetStranKnihy
http://linked.open...cetTvurcuVysledku
http://linked.open...vavai/riv/projekt
http://linked.open...UplatneniVysledku
http://linked.open...iv/tvurceVysledku
  • Vaňková, Radomíra
http://linked.open...n/vavai/riv/zamer
number of pages
http://bibframe.org/vocab/doi
  • 10.1007/978-1-4614-0634-1_19
http://purl.org/ne...btex#hasPublisher
  • Springer-Verlag
https://schema.org/isbn
  • 978-1-4614-0633-4
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