About: Chronic endothelin receptor blockade reduces end-organ damage independently of blood pressure effects in salt-loaded heterozygous Ren-2 transgenic rats     Goto   Sponge   NotDistinct   Permalink

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Description
  • The present study was performed to evaluate the role of an interaction between the endothelin (ET) and the renin-angiotensin systems (RAS) in the development and maintenance of hypertension and in hypertension-associated end-organ damage in heterozygous male and female transgenic rats harboring the mouse Ren-2 renin gene (TGR).
  • The present study was performed to evaluate the role of an interaction between the endothelin (ET) and the renin-angiotensin systems (RAS) in the development and maintenance of hypertension and in hypertension-associated end-organ damage in heterozygous male and female transgenic rats harboring the mouse Ren-2 renin gene (TGR). (en)
  • Chronická blokáda endothelinu redukuje poškození koncových orgánů nezávisle na efektech krevním tlaku u solí přetížených heterozygonních Ren-2 transgenických krys (cs)
Title
  • Chronická blokáda endothelinových receptorů snižuje poškození koncových orgánů nezávisle na vlivu krevního tlaku u solí krmených heterozygotních Ren-2 transgenických krys (cs)
  • Chronic endothelin receptor blockade reduces end-organ damage independently of blood pressure effects in salt-loaded heterozygous Ren-2 transgenic rats
  • Chronic endothelin receptor blockade reduces end-organ damage independently of blood pressure effects in salt-loaded heterozygous Ren-2 transgenic rats (en)
skos:prefLabel
  • Chronická blokáda endothelinových receptorů snižuje poškození koncových orgánů nezávisle na vlivu krevního tlaku u solí krmených heterozygotních Ren-2 transgenických krys (cs)
  • Chronic endothelin receptor blockade reduces end-organ damage independently of blood pressure effects in salt-loaded heterozygous Ren-2 transgenic rats
  • Chronic endothelin receptor blockade reduces end-organ damage independently of blood pressure effects in salt-loaded heterozygous Ren-2 transgenic rats (en)
skos:notation
  • RIV/00216208:11120/04:00001321!RIV09-MZ0-11120___
http://linked.open...avai/riv/aktivita
http://linked.open...avai/riv/aktivity
  • P(LN00A069), P(NA6523), P(NR7826)
http://linked.open...iv/cisloPeriodika
  • 6
http://linked.open...vai/riv/dodaniDat
http://linked.open...aciTvurceVysledku
http://linked.open.../riv/druhVysledku
http://linked.open...iv/duvernostUdaju
http://linked.open...titaPredkladatele
http://linked.open...dnocenehoVysledku
  • 557730
http://linked.open...ai/riv/idVysledku
  • RIV/00216208:11120/04:00001321
http://linked.open...riv/jazykVysledku
http://linked.open.../riv/klicovaSlova
  • hypertension; endothelin system; renin-angiotensin system; bosentan; end-organ damage (en)
http://linked.open.../riv/klicoveSlovo
http://linked.open...odStatuVydavatele
  • CZ - Česká republika
http://linked.open...ontrolniKodProRIV
  • [F130799EB433]
http://linked.open...i/riv/nazevZdroje
  • Physiological Research
http://linked.open...in/vavai/riv/obor
http://linked.open...ichTvurcuVysledku
http://linked.open...cetTvurcuVysledku
http://linked.open...vavai/riv/projekt
http://linked.open...UplatneniVysledku
http://linked.open...v/svazekPeriodika
  • 53
http://linked.open...iv/tvurceVysledku
  • Malý, J.
  • Zima, T.
  • Dvořák, P.
  • Tesař, V.
  • Vernerová, Zdeňka
  • Janda, J.
  • Kopkan, L.
  • Bader, M.
  • Opočenský, M.
  • Vaněčková, I.
  • Kramer, H.
  • Bäcker, A.
  • Ganten, D.
http://linked.open...ain/vavai/riv/wos
  • 000226116600002
issn
  • 0862-8408
number of pages
http://localhost/t...ganizacniJednotka
  • 11120
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