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  • Pain can be considered a specific stressor activating the same brain structures as other non-nociceptive stressors. On the other hand, stress has been found to be a powerful activator of descending antinociceptive system, which is mediated by opioid or non-opioid mechanisms in dependency on the stressor intensity. These pain-modulating networks can cooperate synergically or potentiate themselves. Nevertheless, endogenous pain-modulating circuitry is not fixed; besides an inhibitory control there exists a descending facilitatory system. The final effect of pain modulation depends on a balance between inhibitory and facilitatory inputs. We performed a pilot study testing the modulation of stress and pain by new substance TH-1212. (en)
  • V tomto článku je studován vztah bolesti a stresu. Součástí stresové reakce je aktivace descendentního antinocicepčního systému, který v závislosti na intenzitě tlumí bolest cestou opioidní a neopioidní analgezie. Výsledný efekt modulace bolesti závisí na vzájemném poměru inhibičních a facilitačních vlivů. Pro ovlivnění stresu a bolesti je testována látka TH-1212.
Title
  • Common Mechanisms of Pain and Stress (en)
  • Společné mechanizmy bolesti a stresu
  • Společné mechanizmy bolesti a stresu (cs)
skos:prefLabel
  • Common Mechanisms of Pain and Stress (en)
  • Společné mechanizmy bolesti a stresu
  • Společné mechanizmy bolesti a stresu (cs)
skos:notation
  • RIV/00216208:11120/01:00110060!RIV/2002/MSM/111202/N
http://linked.open.../vavai/riv/strany
  • 18;22
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  • P(LN00B122)
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  • Suppl. 3
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  • 696760
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  • RIV/00216208:11120/01:00110060
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  • Common;Mechanisms;Stress (en)
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  • CZ - Česká republika
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  • [4F1B08F9015D]
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  • Psychiatrie
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  • 5
http://linked.open...iv/tvurceVysledku
  • Pometlová, Marie
  • Rokyta, Richard
  • Yamamotová, Anna
issn
  • 1211-7579
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  • 11120
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